Video: Molecular motors txav li cas?
2024 Tus sau: Miles Stephen | [email protected]. Kawg hloov kho: 2023-12-15 23:36
Tsav cov proteins yog ib chav kawm ntawm molecular motors uas tuaj yeem txav mus los raws li cytoplasm ntawm cov tsiaj cell. Lawv hloov chemical zog rau hauv kev ua haujlwm txhua yam los ntawm hydrolysis ntawm ATP.
Ua li no, molecular motors ua haujlwm li cas?
Molecular motors siv adenosine triphosphate (ATP) hydrolysis rau Lub zog txav ntawm subcellular Cheebtsam, xws li organelles thiab chromosomes, raws li ob lub polarized cytoskeletal fibers: actin filaments thiab microtubules. Tsis muaj lub cev muaj zog paub rau txav mus rau nruab nrab filaments.
Qhov thib ob, puas yog molecular motors enzymes? Molecular motors yog cov enzymes uas hloov chemical zog mus rau hauv mechanical ua hauj lwm. Hauv cytoplasm ntawm eukaryotic hlwb, peb chav sib txawv ntawm lub cev muaj zog uas ua kom muaj kev txav mus los tau paub tias muaj nyob - myosin, kinesin thiab dynein.
Tom qab ntawd, hom molecular motors yog dab tsi?
Muaj peb qhov loj hom molecular motor : microtubule-associated kinesins thiab dyneins, thiab actin-associated myosins.
Dab tsi yog qhov sib txawv ntawm cov motors proteins?
Tsuas yog peb tsev neeg ntawm lub cev muaj zog proteins -myosin, kinesin, thiab dynein-power feem ntau eukaryotic cellular txav (Fig. 36.1 thiab Table 36.1). Thaum lub sij hawm evolution, myosin, kinesin, thiab Ras tsev neeg guanosine triphosphatases (GTPases) zoo li tau sib koom ib tug poj koob yawm txwv (Fig.
Pom zoo:
Dab tsi yog qhov kev txav ntawm nthwv dej piav qhia li cas?
Wave motion, propagation of disruptions - uas yog, deviations los ntawm ib lub xeev ntawm so los yog equilibrium - los ntawm qhov chaw mus rau qhov chaw nyob rau hauv ib tug tsis tu ncua thiab kev koom tes. Cov neeg paub zoo tshaj plaws yog cov nthwv dej saum npoo av, tab sis ob qho tib si suab thiab lub teeb mus ncig raws li kev cuam tshuam zoo li wavelength, thiab cov lus tsa suab ntawm tag nrho cov subatomic particles nthuav tawm wavelike zog
Yuav ua li cas dej txav mus rau cov phab ntsa nyias ntawm ib lub raj tshwj xeeb nws yog dab tsi?
Cov dej molecules tuaj yeem tsim cov hydrogen bonds nrog lwm yam khoom. Txoj kev nyiam ntawm cov dej nce hauv lub raj nyias yog hu ua capillary action. Dej yog attracted rau ntawm phab ntsa ntawm lub raj, thiab cov dej molecules yog attracted rau ib leeg. Lub thinner lub raj, qhov siab dua cov dej yuav nce hauv nws
Vim li cas cov chromosomes ntxiv lossis ploj lawm yuav ua rau cov phenotypes txawv txav?
Ib qho ntxiv lossis tsis muaj chromosome yog ib qho ua rau muaj qee yam kab mob caj ces. Qee cov qog nqaij hlav cancer kuj muaj cov lej txawv txav ntawm chromosomes. Kwv yees li 68% ntawm tib neeg cov qog nqaij hlav yog aneuploid. Aneuploidy originates thaum lub sij hawm cell faib thaum lub chromosomes tsis cais kom zoo ntawm ob lub hlwb (nondisjunction)
Vim li cas qhov thib ob ionization zog ntawm lithium thiaj li txawv txav loj dua thawj?
Thib ob Ionisation Energies yeej ib txwm siab dua thawj zaug vim yog ob qho laj thawj tseem ceeb: Koj tshem tawm cov hluav taws xob los ntawm txoj hauj lwm uas nws ze dua me ntsis rau lub nucleus, thiab yog li ntawd yuav muaj kev nyiam ntau dua rau lub nucleus
Bohr xav li cas txog kev txav ntawm electrons?
Bohr tau hais tias ib qho hluav taws xob pom tsuas yog nyob rau hauv ib qho kev ncig, lossis orbits, nyob ib ncig ntawm lub nucleus. Tus nqi ntawm lub zog ib qho hluav taws xob tau txav los ntawm ib qib zog mus rau lwm qhov. Piav qhov txawv ntawm lub orbit nyob rau hauv tus qauv bohr thiab lub orbital nyob rau hauv lub quantum mechanical qauv ntawm lub atom